Spike Protein–Associated Neuroinflammation: Gliosis and Neuronal Loss in Hippocampal CA1-CA3 and Cerebellum
“Gliosis” and “neuronal cell death” after exposure to a biological agent are core neuropathology endpoints that reflect neuroinflammation, synaptic dysfunction, and impaired neuronal survival. When investigators report gliosis in the hippocampal CA1–CA3 fields and in the cerebellum, they are describing regionally relevant activation of glial cells and downstream neurodegeneration. The hippocampus is critical for declarative… Read More »